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Our model is complementary to the work of [10] since it focuses on the detailed biochemical mechanisms by which of APAP is detoxified in the liver under both normal and overdose situations

350 Mitochondrial dysfunction in Schwann cells triggers alterations in lipid metabolism, leading to myelin sheath dysfunction and accumulation of toxic lipid intermediates, which cause axonal degeneration and neuropathy

Funding Open access funding provided by Karolinska Institute

In addition to axon length, diseased CSMN treated with NMN also displayed improved branching and arborization especially within the 50200 m radius, as revealed by Sholl analyses (Fig