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[DOI] [PubMed] [Google Scholar] 185.Falcone Ferreyra M.L., Emiliani J., Rodriguez E.J., Campos-Bermudez V.A., Grotewold E., Casati P

The only problem is that the inhibitor has a relatively short retention time in the body: its too small and the kidneys would filter it out

In: Proceedings of the National Academy of Sciences, 74(3), S

Of note, neuroinflammation has furthermore been implicated in the pathophysiology of depression [87], whereas increased inflammatory markers, such as C-reactive protein (CRP) or interleukin-2 (IL-2), have been specifically described to be more associated with the atypical subtype of depression [69]

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