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Drake et al., 2017

This range comes from clinical practice observations, not formal FDA trials

The unfolded protein response (UPR) alleviates ER stress through negative feedback mechanisms, which include degrading mRNA, decreasing protein synthesis, inhibiting translation, and posttranslational processes, and facilitating the transport of misfolded proteins to the cytoplasm for degradation [47]

Identifying metabolic changes specific to the deletion of GDH2 and GDH3 genes will assist in the characterisation of the role of glutamate dehydrogenase in C

Our findings suggest that the novel hub gene GPX3 is involved in the neutrophil-driven oxidative stress-mediated pathogenesis of RA

Some researchers tolerate dose increases faster