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Nrf2 and oxidative stress: a general overview of mechanisms and implications in human disease

PKC/ZFP64/CSF1 axis resets the tumor microenvironment and fuels anti-PD1 resistance in hepatocellular carcinoma

2.2.2 Pre-clinical studies Preclinical studies have provided further insights into the potential benefits of SF in schizophrenia

However, under oxidative stress, oxidative modification of cysteine residues in Keap1 causes conformational changes that weaken its binding to Nrf2, allowing Nrf2 to dissociate from Keap-1 and rapidly translocate to the nucleus

Overexpression of CHAC1 in prostate cancer cells increased intracellular lipid peroxidation and decreased levels of GPX4, promoting ferroptosis and enhancing sensitivity to docetaxel