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Post-mortem findings showed higher GSH levels in the caudate, cerebellar, and frontal regions (Tong et al., 2016)

Experiments with a hypoxia tolerant bivalve

These systems may prevent quinones/hydroquinones from redox cycling reactions, the formation of lipid peroxidation products, GSH depletion, and the inactivation of signaling molecules such as hydroxynonenal and other molecules derived from the lipid peroxidation process capable of triggering apoptosis and ferroptosis (Dodson et al

The result is improved tissue health, faster recovery, and a renewed capacity for regeneration, offering hope for age-related conditions (4)

Excited-state intramolecular proton-transfer (ESIPT)-inspired solid state emitters

A degradative to secretory autophagy switch mediates mitochondria clearance in the absence of the mATG8-conjugation machinery