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In summary, AD is accompanied by a distinct gut microbiome signature that likely contributes to disease via increased inflammation, reduced neuroprotective metabolites, and possibly direct effects on protein pathology

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We also found that the levels of PG0006550 and PG0005794 genes related to IAA were significantly downregulated after treatment

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CHAC1s contribution to tumor progression is markedly influenced by the cellular and tumor context, differing across various cancer types