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Data are normalized to the NAD + abundance

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To view digital features for this article, go to Introduction For several decades, N -acetylcysteine (NAC) has been used in clinical practice worldwide in a variety of indications: as a mucolytic, in acetaminophen intoxication, doxorubicin-induced cardiotoxicity and in other cardiovascular, respiratory and psychiatric disorders [1]

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Unfortunately, the application of BRAF inhibitors vemurafenib, dabrafenib, and encorafenib has failed in KRAS driven tumors, because of the paradoxical activation of ERK1/2