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However, it should be acknowledged that current studies on the single mechanism of PSP repair of the intestinal mucosal barrier have focused mainly on the repair of the intestinal mucosal barrier by SCFA, a metabolite of the regulatory flora

Makes it susceptible to rapid enzymatic degradation by peptidases in serum and tissues

This modification results in dramatically different properties

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In contrast, the Ph HAT dimeric interface is exclusively formed by -helical interactions, and, under this arrangement, the 1 strands are pushed away from the dimerisation interface (Supplementary Fig