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Activity-based probes developed by applying a sequential dehydroalanine formation strategy to expressed proteins reveal a potential -globin-modulating deubiquitinase

Such damage can further lead to mitochondrial dysfunction, triggering apoptosis or necroptosis, which represents a key pathological process in oxidative stress [61]

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Ramadan et al., 2022

Whether the mitochondrial dysfunction in RA can be associated with ferroptosis and what kind of ferroptotic cells should be linked to the mitochondrial dysfunction in RA still need further research and exploration

Cancer Cell 2004