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Several mechanisms have been proposed to explain chronic hyperglycemia-induced nerve damage, such as intracellular accumulation of sorbitol, glycation reactions, and oxidative and nitrosative stress (reviewed in 69)

Common mild effects: Temporary flushing or warmth at infusion site (typically brief) Mild headache in some patients at higher doses Rare nausea Important clinical considerations: Patients with asthma should be evaluated prior to IV glutathione because inhaled glutathione has been associated with bronchospasm in some asthmatic individuals

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(d) Neuroprotective: Some studies on BPC-157 revolve around nerve support and protection

The factors that lead to depletion of glutathione includes natural ageing process, or the external stressors that include chronic exposure to chemical toxins, cadmium exposure, alcohol use, smoking, pollution, strenuous aerobic activities, dietary factors, ultraviolet (UV) radiation exposure, tissue injury as from burns, ischemia, septic shock, surgery, drugs (such as acetaminophen and adriamycin)

We dont recommend that approach and the reasons are specific, not just liability-driven