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While early efforts focused on introducing defensins to combat phytopathogenic fungi (Wang et al., 1999

They generate the vast majority of our cellular energy in the form of adenosine triphosphate (ATP)

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Inflammatory cytokines and enzymes, such as interleukin-6 (IL-6), interleukin-17 (IL-17), tumor necrosis factor-alpha (TNF-), nitric oxide (NO), inducible nitric oxide synthase (iNOS), and matrix metalloproteinases (MMPs), show a high correlation with the pathogenesis of hemorrhoids

In cells, oxidative stress generated by Reactivating p53 and Inducing Tumor Apoptosis (RITA) induces TrxR1 tetramerization and covalent linkage, resulting in reduced activity or inactivation of TrxR1 (Xu et al., 2015)