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The mechanistic role of oxidative stress in viral replication is through the induction of host transcriptional factor Hypoxia-Inducible Factor-1 (HIF-1) by the HIV protein Vpr to increase mitochondrial activity, resulting in ROS production which activates HIV-1 LTR [22,23]

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Initial benefits of RT primarily begin at the molecular level, as even an acute bout of resistance exercise reduces inflammation and increases antioxidant defenses (zbeyli et al., 2017

These included structural features (bond lengths and angles), frontier orbital energies (EHOMO and ELUMO), and the derived energy gap that governs intramolecular charge transfer processes

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Importantly, the multi-system functional improvements observed strongly imply successful engraftment or sustained bioactivity