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Crosslink between mutations in mitochondrial genes and brain disorders: implications for mitochondrial-targeted therapeutic interventions

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Neurotrophins and schizophrenia

Finally, we see clear priorities for the field: consensus SOPs and reference materials, routine compartment/time stamps in datasets, better in-vivo iron/ferroptosis assessment and mechanism-anchored, biomarker-guided clinical trials

[DOI] [PubMed] [Google Scholar] 88.Langer H.T., Ramsamooj S., Liang R.J., Grover R., Hwang S.K., Goncalves M.D.S

288 MEK activation further induces double phosphorylation of a critical TPY motif of p38 on tyrosine and threonine residues