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16,22-24 Tezacaftor/ivacaftor is approved for individuals aged 6 years and older with a homozygous F508del mutation or one tezacaftor/ivacaftor-responsive CFTR mutation

In brief, inhibition of TrxR augments the susceptibility of tumor cells to oxidative stress, bolsters the selective eradication of cancerous cells, and instigates the cascade of apoptosis, which has received widespread attention in recent years making TrxR a potential target for cancer therapy (191, 192)