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doi: 10.3389/fmolb.2022.858142 Received 19 January 2022 Accepted 04 April 2022 Published 04 May 2022 Volume 9 - 2022 Edited by Antentor Hinton, Vanderbilt University, United States Reviewed by Jan Riemer, University of Cologne, Germany Ester Zito, Mario Negri Pharmacological Research Institute (IRCCS), Italy Updates Copyright 2022 Gansemer and Rutkowski

A broader analysis revealed over 2,700 regions with reduced methylation in cancer and over 1,500 regions that curcumin remethylated, highlighting its ability to restore a more normal epigenetic state ( Figure 1 To assess the impact of curcumin on DNA methylation, another study used three CRC cell linesa harbor cancer therapy group cell line 116 (HCT116), a human tumor cell line 29 (HT29), and RKOwhich were exposed to curcumin treatment ( Epithelialmesenchymal transition (EMT) is a key process in CRC progression and metastasis, characterized by the loss of epithelial markers (e.g., E-cadherin) and the gain of mesenchymal markers (e.g., vimentin and ZEB1), enhancing cancer cell migration and invasion ( 3.1.3 Histone modification effects Recent research also shows that histone modifications can be regulated by CUR (Figure 1)

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