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Recent evidence further indicates that H3K14la contributes to neuronal ferroptosis after intracerebral hemorrhage by upregulating PMCA2 and impairing calcium efflux, thereby aggravating neural injury [90]

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targeted NNMT knockdown protected against DIO by increasing energy expenditure

In vivo efficacy of ATO+MTX against primary ALL Cryopreserved primary ALL cells obtained from a mouse of the IL7R knock-in model (43) or from 4 pediatric ALL patients (2 BCP- and 2 T-ALL) were used in this study