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L-Lysine also contributes to energy production when converted to acetyl coenzyme A, one of the principal fuels for the Krebs cycle

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10.1016/j.ejca.2018.03.012 Summary Keywords mTORC1, mitochondria, mitochondrial functions, cancer, therapy Citation de la Cruz Lpez KG, Toledo Guzmn ME, Snchez EO and Garca Carranc A (2019) mTORC1 as a Regulator of Mitochondrial Functions and a Therapeutic Target in Cancer

Under hypoxia, cell metabolism shifts away from mitochondrial OXPHOS to glycolysis for energy production, which is primarily mediated by activation of HIF-1 signaling

suggests these actions might involve the activation of hormone-sensitive lipase (HSL), an enzyme critical for hydrolyzing stored triglycerides into free fatty acids and glycerol, possibly mitigating adipocyte size