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Multiple residues in DET1 form hydrogen bonds and electrostatic interactions with Ube2e2, including DET1 D289 : Ube2e2 R126 , DET1 H297 : Ube2e2 T201 , DET1 D324 : Ube2e2 R88 and Ube2e2 R124 , DET1 R317 : Ube2e2 T68 (Fig

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These mediators promote tumor cell adaptation to hypoxia and nutrient deprivation, thus facilitating tumor survival and growth [137, 138]

We perform untargeted metabolomics on glucose-avid non-small cell lung cancer (NSCLC) mouse models of human cancer and find that accumulation of reduced glutathione is prominent in tumors relative to normal lung

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