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Because prostaglandins and angiotensins are used to expand the afferent arteries, anti-prostaglandin drugs such as nonsteroidal anti-inflammatory drugs (NSAIDs), drugs that have anti angiotensin activity such as angiotensin-converting enzyme inhibitors (ACEIs), or angiotensin receptor blockers (ARBs) can cause nephrotoxicity in the glomerulus Tubular cell toxicity: tubular cells are exposed to a toxic substance (nephrotoxin)

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These cells were also positive for human albumin (Fig

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MotionCor2: anisotropic correction of beam-induced motion for improved cryo-electron microscopy

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It is unclear whether FoxO3, TLX and REST act in concert or function as distinct pathways in regulating adult neurogenesis

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