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Eryptosis inhibitors A growing body of evidence from ex vivo experiments and animal models highlights the role of biomolecules or synthetic compounds, whether from dietary sources or administered as pharmaceuticals, respectively, in mitigating eryptosis (Table S5)

The researchers proposed that the compounds effects on muscle function operate through boosting NAD+ levels by inhibiting NNMTs consumption of nicotinamide, more substrate is available for NAD+ synthesis via the salvage pathway, supporting mitochondrial function and energy metabolism in aged skeletal muscle

CLA has been extensively studied for its ability to support normal cholesterol levels and promote a healthy metabolic rate

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The notion that oxidative damage likely influences konzo susceptibility has been long theorized since this disease is associated with the consumption of cyanogenic compounds that are well known to increase radicals either through direct inhibition of mitochondrial respiration or through toxicity of the downstream metabolites of cyanide 12,18

A secondary planned outcome assessment of changes in LDL density, registered on clinicaltrials.gov, was not assessed due to the underdeveloped experimental nature of the assay