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Bassett JHD, Williams GR

But hope was kindled when, within a description of one of their mouse studies, I came across this promising line: Of note, the increase in atherosclerotic plaque burden with dietary L-carnitine occurred in the absence of proatherogenic changes in plasma lipid, lipoprotein, glucose, or insulin levels. Excellent, they acknowledged glucose and insulin as potential risk factors

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The incidence of drug-related adverse events , mainly mild gastrointestinal and obstetrical complications, was slightly more frequent in the placebo arm (lactose intolerance) and resolved rapidly (Table 1B)

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