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Erastin can interact with VDAC2 protein to trigger mitochondrial malfunction and oxidative substance release, eventually leading to oxidative cell death, i.e., ferroptosis (53) (Figure 1)

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Neutrophil adaptations upon recruitment to the lung: new concepts and implications for homeostasis and disease

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Consistent with their elevated SLC7A11 expression level, IRE1-null MDA-MB-231 cells also demonstrated increased cystine uptake, which can be inhibited by IRE1 reconstitution (Supplementary Fig