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This conservative approach minimizes copper toxicity risk but may require 16+ weeks to produce observable density changes

Trauma, shock, and gut translocation

The biochemical basis for cobalamin neuropathy remains obscure

showed that during intestinal inflammation, the GVB is disrupted, thus determining a state of bacterial product translocation and systemic inflammation, eventually leading to the propagation of inflammation to the brain, while the vascular barrier in the choroid plexus shuts down, helping protect the brain from inflammation but also potentially impairing some brain functions and causing anxiety [42]

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