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Semaglutide provides the core metabolic benefits: appetite suppression, improved insulin sensitivity, reduced glucagon secretion, and delayed gastric emptying

As ferroptosis is driven by lipid peroxidation and oxidative stress, xCT sustains glioblastoma survival by maintaining intracellular cysteine levels, which serve as a precursor for GSH synthesis, a key antioxidant that mitigates ROS-induced lipid peroxidation [49,50,51]

Ferroptosis induction was coupled to an increase in turnover of LC3B and autophagosome formation, consistent with the notion that lipid peroxidation as well as oxidized lipids could promote autophagy activation [63]

10.1007/s11095-024-03771-6 [DOI] [PubMed] [Google Scholar] Jastreboff, A

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