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Description

An alternative NF-B pathway is activated by TNF-family cytokineslymphotoxin b (TNFSF3) CD40 ligand (CD40L and TNFSF5), B cell activating factor (BAFF and TNFSF13B), and receptor activator of NF-B ligand (RANKL and TNFSF11)

These results establish CitAgNPs as a versatile and safe therapeutic option for managing resistant Candida infections, advancing antifungal therapy with novel, application-specific solutions

Mechanistically, the expression of GLS1 is regulated indirectly by Myc (via repression of miR-23a and miR-23b) and mTORC1 11,12

LDL binding to the LOX-1 receptor increased OS, decreased NO production, potentiated superoxide generation, and activated NF-kB-all of which exacerbate DM pathogenesis