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Elevated expression of IGF-IR in BCR/ABL + cells may promote the development and self-renewal of chronic myeloid leukemia, while downregulation of IGF-IR in BCR/ABL + cells leads to acute lymphoblastic leukemia (ALL) [14]

To make the problem tractable, they made a couple of (fairly heroic) simplifying assumptions: first that all the activation gates were shut at the resting potential (m 0 = 0), and second that at steady-state there was complete inactivation at the clamp potential (h = 0)

This research was funded by Undergraduate Innovation and Entrepreneurship Training Program of Jiangsu Province (202310304155Y)

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Once neuroplasticity is established (after the induction phase), less frequent maintenance dosing may sustain effects, but initial protocols should follow daily administration schedules to maximize therapeutic outcomes

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