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doi: 10.1016/j.yexcr.2020.112362

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GDF11 contributes to hepatic hepcidin (HAMP) inhibition through SMURF1-mediated BMP-SMAD signalling suppression

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Since depletion of GSH has been implicated in the etiology and pathogenesis of cataracts and other eye disorders, a logical approach to drug development would be the prevention of oxidative damage to the lens by the use of GSH prodrugs