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Interferon- and ATRA are important mediators for the upregulation of CD38 in NSCLC cells, which allows the tumor to develop resistance to anti-PD-L1 and anti-PD-1 treatments

(50) reported that sepiapterin, a tetrahydrobiopterin precursor, recoupled NOS in CRC cell lines (HCT116 and HT29), restoring the tetrahydrobiopterin: dihydrobiopterin ratio, which is significantly lower in tumors than in normal tissues

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