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In the context of renal injury, GM-CSF, LPS, IL-1, TNF-, FN-, and various endogenous damage-associated molecular patterns (DAMPs) released by the damaged renal cells promote the polarization of macrophages to the M1 phenotype ( The number of M2 macrophages is decreased in the early and middle stages of renal injury, whereas it is increased in the end-stage ( Although M2 macrophages contribute to tissue repair, they can also promote fibrosis

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