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& Weissberger, A

632 Di-Huang-Yin-Zi effectively decreases ROS and MDA levels in prefrontal cortex, increases the expression of ferroptosis-related markers GPX4 and SLC7A11 and, and decreases iron deposition, thereby improving depressive symptoms and synaptic ultrastructure of cortical regions in post-stroke depressed (PSD) rats, suggesting that Di-Huang-Yin-Zi can reduce the symptoms and enhance function in PSD rats by inhibiting ferroptosis through SLC7A11/GPX4 pathway

Inhibition of ACSL4 has been shown to protect cells from ferroptosis

When membranes function properly, they can efficiently eliminate toxins while protecting cellular contents

We calculated the effect size as a standardized mean difference of the final scores and summarized it using a Hartung-Knapp-Sidik-Jonkman (HKSJ) random effects meta-analysis, and we switched to the DerSimonian-Laird random effects model if meta-analysis results from HKSJ were counter-intuitive