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Likewise in our previous studies of glaucoma, we have observed metabolic deficiency in the DBA/2J optic nerve that coincides with an early period of degeneration [69]

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Kyoto Encyclopedia of Genes and Genomes (KEGG)-based analysis showed that the differentially expressed metabolites were involved in arachidonic acid metabolism, the glycerophospholipid pathway, and inflammatory mediator regulation of Transient receptor potential channel (TRP) channels, suggesting that allicin may change the metabolism in the peri-infarct cortex, especially arachidonic acid metabolism (Fig

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In particular, astrocytes, as the cells supporting neuronal function, seem to be of great importance for our understanding of underlying disease mechanisms (5)

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